Morphological/nanostructural control toward intrinsically stretchable organic electronics - Chemical Society Reviews (RSC Publishing) DOI:10.1039/C8CS00834E
Interleukin-1 alpha and high mobility group box-1 secretion in polyinosinic:polycytidylic-induced colorectal cancer cells occur via RIPK1-dependent mechanism and participate in tumourigenesis | SpringerLink
Comanche TX Real Estate - Comanche TX Homes For Sale | Zillow
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Electronic Muscles and Skins: A Review of Soft Sensors and Actuators | Chemical Reviews
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Interleukin-1 alpha and high mobility group box-1 secretion in polyinosinic:polycytidylic-induced colorectal cancer cells occur via RIPK1-dependent mechanism and participate in tumourigenesis | SpringerLink
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If a tree falls in the forest: terrestrial habitat loss predicts caddisfly (Insecta: Trichoptera) assemblages and functional feeding group biomass throughout rivers of the North-central United States | SpringerLink
RNA-binding protein altered expression and mislocalization in MS
Interleukin-1 alpha and high mobility group box-1 secretion in polyinosinic:polycytidylic-induced colorectal cancer cells occur via RIPK1-dependent mechanism and participate in tumourigenesis | SpringerLink
Quantifying and Mitigating Motor Phenotypes Induced by Antisense Oligonucleotides in the Central Nervous System | bioRxiv
Interleukin-1 alpha and high mobility group box-1 secretion in polyinosinic:polycytidylic-induced colorectal cancer cells occur via RIPK1-dependent mechanism and participate in tumourigenesis | SpringerLink
Pharmaceutics | Free Full-Text | Safe and Effective Cynomolgus Monkey GLP—Tox Study with Repetitive Intrathecal Application of a TGFBR2 Targeting LNA-Gapmer Antisense Oligonucleotide as Treatment Candidate for Neurodegenerative Disorders | HTML
Arbidol inhibits human esophageal squamous cell carcinoma growth in vitro and in vivo through suppressing ataxia telangiectasia and Rad3-related protein kinase | eLife
RNA-binding protein altered expression and mislocalization in multiple sclerosis | bioRxiv
PDI is an essential redox-sensitive activator of PERK during the unfolded protein response (UPR). - Abstract - Europe PMC
Pharmacological induction of selective endoplasmic reticulum retention as a strategy for cancer therapy | Nature Communications